264256128>2048141620.250.250.1250.516bladder showed that rough and coarse mucosa at the fundus with marked wall thickness and micro-scopic examination showed infiltration of bile containing foam cells along with lymphocytes (Figure 1D-F). Gram staining showed no evident bacterial colonies, although some neutrophilic reac-tions were observed, histologically. The patient was pathologically diagnosed with focal acute inflammatory phase XGC. S. capitis was isolated Drug susceptibility profiles of A. baumanniiDrugsABKAMKAZTCAZIPMMPMCIPCSTPIP*ABK, arbekacin; AMK, amikacin; AZT, aztreonam; CAZ, ceftazidime; IPM, imipenem; MPM, meropenem; CIP, ciprofloxacin; CST, colistin; PIP, piperacillinTable 2 MIC and susceptibility of isolated bacteria to antibioticsFigure 2 Relative abundance of major bacteria in bileMICs in mg/Lfrom the bile sample obtained from Patient 2 during the operation, with this isolate being resistant to ABPC, CAZ, EM, GM, LVFX, OXA, and PCG, but susceptible to ABK, TEIC, and VCM (Table 2). Metagenomic analysis of the bile sample from Patient 2 showed bacterial DNA derived from four genera, with the genus Staphylococcus being predominant. Filtering of the data sets revealed bacterial DNA from two phyla, Firmicutes (97%) Drug susceptibility profiles of S. capitisDrugs*ABKABPCCAZCEZEMGMLVFXOXAPCGTEICVCM*ABK, arbekacin; ABPC, aminobenzylpenicillin; CAZ, ceftazidime; CEZ, cefazolin; EM, erythromycin; GM, gentamicin; LVFX, levofloxacin; OXA, oxacillin; PCG, penicillin G; TEIC, teicoplanin; VCM, vancomycinMICs in mg/L645121024640.251285
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